The above question is one we answer every day. And no matter how many times we repeat our answer (many times it’s even woven into our announcement of a new title), people still ask. So here’s the answer:
We don’t know. Our individual retailers get to decide which Lost Art Press books they carry and which they don’t.
Plus, every retailer orders books on different schedules, so we have no idea if Lee Valley or Dictum is going to carry a book until months later when they issue a purchase order.
Our recommendation: Send a note to your local retailer asking if they are going to carry the title you are interested in. Chances are if enough people ask about the book, they will carry it.
The Ordering Process
For those of you who want all the details, here is how we sell books to retailers. When a book is released, Lost Art Press is the sole source for that title for 30 days. This gives us a small headstart on selling our own title to our regular customers.
After 30 days, the book is added to our wholesale price list and sent out to our retailers. Some of them want to see the physical book before they make a decision. Others want to see a pdf of the book to make sure the content is something for their audience. Still others order it sight-unseen.
Most of our retailers place a few big orders during the year instead of ordering a big batch of a single book. This saves them on shipping charges. So they might order stock for Christmas at the end of the summer and restock in January. Other retailers order books when they are released. Still other retailers might wait a year to see if there is demand for it in their market before ordering.
After they place the order, we assemble a pallet of the things they order and we await a truck to pick them up. The truck’s schedule is usually out of our hands. Sometimes it can take weeks for a truck to show up.
After the retailer receives the new book, they usually write their own product description that suits their audience. So they have to read (or skim) the book, take photos, update their website etc.
All this takes time and – as I said – is completely out of our hands.
This story tells how 30,000 rosewood logs were illegally harvested in Madagascar and trafficked on a cargo ship to Singapore. And why they are now sitting in a warehouse, attracting termites.
According to the University of Oxford, “Timber harvested from rosewoods has been the world’s most trafficked wild product since 2005, accounting for 30-40% of the global illegal wildlife trade (more than all animal products put together).”
As I got ready to teach my Dutch tool chest class last weekend, I realized I was incredibly rusty as far as succinct instruction for this project. I checked my calendar, and I’m pretty sure the last time I taught this class in our shop was the weekend of May 6, 2022 – and the last time I taught it at all was at the Florida School of Woodwork in October of 2022. So it’s been two years since last I tried to cram all the instruction into just a few days.
Instead, I was busy expounding and expanding instruction in “Dutch Tool Chests,” which (if the printer deities are smiling upon me) will be in our warehouse in early November (and we’ll offer a pdf free with purchase for the first 30 days it’s on sale, just FYI, since a couple people have asked). In the book, I offer a brain dump on different ways to approach the various building steps, so that readers can choose what works best for their mindset and tool set.
Take rabbets, for example, which are cut to create a raised panel on the fall front and chest lid (and as practice for cutting a square rabbet in a low-risk decorative situation, where it’s OK if it’s slightly out of square!).
In the book, I mention the dado stack for those who liked a tailed approach, then cover at some length techniques for cutting a crisp rabbet with a skew rabbet plane, a straight rabbet plane or a shoulder plane (there are other hand-tool ways of course – but I teach what I know best).
In a short class, though, it simply isn’t possible to explore the options and offer choices/decisions. Instead I sharpen and set up ahead of time our Veritas skew rabbet planes (which are technically moving fillisters) for a 3/4-wide x 1/16″ deep cut, show the students how to use the tool (preferably without cutting myself on the corner of the blade while looking up and talking about the work instead of paying proper attention to what I’m doing), then send them off to do the work.
After filling almost 200 pages of “here’s lots of options,” it was difficult to remember to dial it back in the shop to what was possible to achieve in three days, with limited tool sets and varying skill sets among the students. But thank goodness I dusted off my in-person DTC teaching skills last weekend in our shop, where I know where to find everything. The next time I teach this tool chest will be in a few weeks in England, at the London IWF, where my own tool set will be severely limited and I’ll have no idea where to find anything.
And by the time I get back from England, I’m hopeful my book will be in the warehouse. Now I just have to not cut my signing fingers (I won’t – it’s always my left hand that gets cut by not paying attention to that pesky pokey-outie skew rabbet blade).
The following is excerpted from Dr. Jeffery Hill’s “Workshop Wound Care.” The book delves right to the heart of what you need to know when faced with common workshop injuries, from lacerations, to puncture wounds to material in the eye. Dr. Hill is an emergency room physician and an active woodworker. So he knows exactly the information a woodworker needs to know when it comes to injuries. And he presents information in a way that a non-medical professional can easily understand it.
There are a number of diseases that, due to their being very uncommonly encountered in the modern world, seem like quaint relics of antiquity. Diphtheria? Rubella? Mumps? Rabies? What even are those? There are a few diseases that have been completely eradicated (smallpox) or nearly completely eradicated (polio) thanks to sustained vaccination efforts. Tetanus, however, is here to stay on this earth through all of eternity regardless of our efforts at vaccination.
Why? Spores, that’s why
What is Tetanus?
Tetanus is a clinical condition caused by the tetanus toxin which produced by the bacteria Clostridium tetani. Clostridium tetani possesses the relatively uncommon ability to form spores (examples of other spore-forming bacteria include Clostridium botulinum, a.k.a. botulism, and Bacillus anthracis, a.k.a. anthrax).
Spores are exceptionally hardy bits of microbiology. Composed of a hard shell and just enough reproductive matter, they are typically produced when the bacteria run into rough times. A lack of nutrients, which would typically just kill off less fastidious bacteria, triggers C. tetani to produce these spores that can survive a lack of water, nutrients, presence of high amounts of radiation, freezing weather, boiling temps and even chemical disinfectants. Spores can remain viable in inhospitable environments for tens of thousands of years. C. tetani spores are most commonly found in soil, dust and manure, but can be found anywhere in the environment.
When tetanus spores find their way back to a hospitable environment they come alive and start to replicate, along the way producing tetanus toxin (awesomely named tetanospasmin). The tetanus toxin is taken up by the nervous system, ultimately ending up in the spinal cord and brain where it acts to block inhibitory signaling pathways. Because two negatives make a positive, the end result of this action is an excess of electrical transmission from the central nervous system to the muscles and severe muscle spasms.
These unopposed muscle contractions lead to the characteristic clinical manifestations of tetanus. “Lock jaw” is due to contraction of the jaw muscles. Contraction of the facial muscles results in “risus sardonicus,” a fixed smile/facial expression. And contraction of the back muscles results in severe arching of the back. But the neurotoxin doesn’t limit itself to the motor system; it can also lead to seizures and uncontrolledblood pressure (both high and low) and heart rate (also high and low).
If you have ever had a charley horse where your legs cramp up, you have a little taste of how terrifically painful muscle spasms can be. Now imagine that affecting your entire body. Also, because the tetanus toxin irreversibly binds with nerve cells, the uncontrolled nerve signals and muscle spasms continue until the body can grow new nerve endings (a process that is weeks to months long). Generalized tetanus can lead to broken bones, spasm of the respiratory muscles, and aspiration of stomach contents and food into the lungs. Ten to 20 percent of patients still die of tetanus despite modern medical therapies.
What Wounds are at High Risk for Tetanus Infection?
Tetanus infections don’t just come from rusty nails. In fact, all wounds – cuts, abrasions (both to the skin and eyes) and burns – are susceptible to tetanus infection. There are certain wounds, however, that are more prone to tetanus infection and create an environment where the tetanus bacteria will produce the tetanus toxin. Wounds that are dirty, puncture-type wounds and crush injuries are most at risk.
Logically, the more tetanus bacteria present in a wound, the higher the risk of infection. As such, injuries with significant contamination with soil are more likely to result in infection.
If the tetanus bacteria is pushed deep into the tissue, infection is more likely. Puncture wounds, in general, carry a higher risk of all types of wound infection. The narrow tract of a puncture wound is apt to quickly close over bacteria and other matter pushed deeply into the tissues. And, it is much more difficult to adequately clean at the time of injury, meaning it is much more difficult to use irrigation to decrease the number of bacteria present.
Because tetanus bacteria are more likely to grow and produce toxin in “devitalized” tissue, crush injuries are also highly susceptible to infection.
How does this translate to you, the woodworker? A scratch or minor cut from a chisel or knife is less risky. A injury from an axe or froe while breaking down green wood is more risky. (Though to reiterate: Any break in the skin carries a risk of tetanus infection.)
What About the Tetanus Vaccine?
The tetanus vaccine includes a component of the tetanus toxin called a toxoid. This toxoid is coupled with a diphtheria toxoid (Td) and often an acellular version of pertussis (TdaP).
As a side note, the risks of contracting diphtheria and pertussis in adulthood are sufficient enough that the administration of the whole package of TdaP is recommended by the CDC. Pertussis (whooping cough) can be common in older adults (often presenting as mild illness) and easily spread to incompletely vaccinated infants (often presenting as a severe respiratory illness). For this reason, if you are going to be around a new baby in the family (e.g. new grandparents), you’ll likely be asked to get a “tetanus booster” (though really you are boosting your pertussis immunity). Back to tetanus…
As with other vaccines, introducing the tetanus toxoid gives the body’s immune system a “wanted” poster of what the real toxin looks like, allowing the body to create neutralizing antibodies to the toxin. The typical vaccination schedule is for three doses of the vaccine to be administered at two months, four months, and six months of age, with boosters given around six to 12 months after the third dose, and again before entering kindergarten. Following that, boosters are recommended every 10 years. In the developed world, adherence to the recommended vaccination schedule is (thankfully) high, meaning that most people are “fully vaccinated” for tetanus through their childhood. It is not uncommon, however, for adults to miss out on their regular boosters. As a result of this, most cases of tetanus are reported in patients 50 years of age and older.
Every chair class Chris teaches seems to develop its own gravitational pull. It’s inevitable – if you orbit within 50 feet of a class taught by Chris Schwarz you will get sucked in.
Now getting sucked into a class can mean many different things: Perhaps it means assisting students taper chair legs by hand until your shirt starts sticking to you; or remaking an arm in record time due to a irreparable and untimely break; or sometimes becoming the designated lunch fetcher of the day (this task can quickly make you popular among the students).
I of course am speaking from experience. I’ve completed all of these tasks at least once and am in no way complaining about it. I openly love the infectious energy of a class week – it’s chaotic and exhausting at times and I live for it. I love walking into work unsure of what the day will hold.
While the palpable energy from last week’s class was no different, one of the tasks I was given was new to me.
With how often we use our tools here, on top of student usage, shop maintenance is a constant. Planes and chisels need sharpening, floors need sweeping and carver’s vises need new jaws.
This is nothing against our beloved carver’s vises; we use them daily here. There is, however, one fault we’ve found with most of them – the softwood jaws that come standard on the vises have a tendency to lose their heartiness over time. (The newly recast Grizzly vise comes with hardwood jaws.)
Either the constant use causes the screw holes of the jaws to strip (which is what happened in this case) or the soft pine cracks. Or both (which is what happened in this case). When these things happen, either oak or hickory are what we typically use for new jaws.
After Chris gave me a quick lesson on how to replace the old jaws, I got to work.
Below is a visual step-by-step of how I made this repair.